128 Mott St # 138 Suite 608
New York NY 10013
Medical School: University Of Connecticut School Of Medicine - 1999
Accepts Medicare: Yes
Participates In eRX: Yes
Participates In PQRS: Yes
Participates In EHR: No
License #: 220066
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Awards & Recognitions
|HCPCS Code||Description||Average Price||Average Price
Allowed By Medicare
|HCPCS Code:69145||Description:Remove ear canal lesion(s)||Average Price:$794.62||Average Price Allowed
|HCPCS Code:31575||Description:Diagnostic laryngoscopy||Average Price:$378.06||Average Price Allowed
|HCPCS Code:31231||Description:Nasal endoscopy dx||Average Price:$441.25||Average Price Allowed
|HCPCS Code:99203||Description:Office/outpatient visit new||Average Price:$287.62||Average Price Allowed
|HCPCS Code:69210||Description:Remove impacted ear wax||Average Price:$148.91||Average Price Allowed
|HCPCS Code:99213||Description:Office/outpatient visit est||Average Price:$160.39||Average Price Allowed
|HCPCS Code:99212||Description:Office/outpatient visit est||Average Price:$127.45||Average Price Allowed
|HCPCS Code:92557||Description:Comprehensive hearing test||Average Price:$111.72||Average Price Allowed
HCPCS Code Definitions
- Comprehensive audiometry threshold evaluation and speech recognition (92553 and 92556 combined)
- Removal impacted cerumen requiring instrumentation, unilateral
- Excision soft tissue lesion, external auditory canal
- Laryngoscopy, flexible fiberoptic; diagnostic
- Nasal endoscopy, diagnostic, unilateral or bilateral (separate procedure)
- Office or other outpatient visit for the evaluation and management of an established patient, which requires at least 2 of these 3 key components: A problem focused history; A problem focused examination; Straightforward medical decision making. Counseling and/or coordination of care with other physicians, other qualified health care professionals, or agencies are provided consistent with the nature of the problem(s) and the patient's and/or family's needs. Usually, the presenting problem(s) are self limited or minor. Typically, 10 minutes are spent face-to-face with the patient and/or family.
- Office or other outpatient visit for the evaluation and management of a new patient, which requires these 3 key components: A detailed history; A detailed examination; Medical decision making of low complexity. Counseling and/or coordination of care with other physicians, other qualified health care professionals, or agencies are provided consistent with the nature of the problem(s) and the patient's and/or family's needs. Usually, the presenting problem(s) are of moderate severity. Typically, 30 minutes are spent face-to-face with the patient and/or family.
- Office or other outpatient visit for the evaluation and management of an established patient, which requires at least 2 of these 3 key components: An expanded problem focused history; An expanded problem focused examination; Medical decision making of low complexity. Counseling and coordination of care with other physicians, other qualified health care professionals, or agencies are provided consistent with the nature of the problem(s) and the patient's and/or family's needs. Usually, the presenting problem(s) are of low to moderate severity. Typically, 15 minutes are spent face-to-face with the patient and/or family.
Medical Malpractice Cases
Medical Board Sanctions
Cardiovascular Disease (Cardiology)
Physical Medicine And Rehabilitation
*These referrals represent the top 10 that Dr. He has made to other doctors
Deletion of FADD in macrophages and granulocytes results in RIP3- and MyD88-dependent systemic inflammation. - PloS one
Myeloid cells, which include monocytes, macrophages, and granulocytes, are important innate immune cells, but the mechanism and downstream effect of their cell death on the immune system is not completely clear. Necroptosis is an alternate form of cell death that can be triggered when death receptor-mediated apoptosis is blocked, for example, in stimulated Fas-associated Death Domain (FADD) deficient cells. We report here that mice deficient for FADD in myeloid cells (mFADD-/-) exhibit systemic inflammation with elevated inflammatory cytokines and increased levels of myeloid and B cell populations while their dendritic and T cell numbers are normal. These phenotypes were abolished when RIP3 deficiency was introduced, suggesting that systemic inflammation is caused by RIP3-dependent necroptotic and/or inflammatory activity. We further found that loss of MyD88 can rescue the systemic inflammation observed in these mice. These phenotypes are surprisingly similar to that of dendritic cell (DC)-specific FADD deficient mice with the exception that DC numbers are normal in mFADD-/- mice. Together these data support the notion that innate immune cells are constantly being stimulated through the MyD88-dependent pathway and aberrations in their cell death machinery can result in systemic effects on the immune system.
Commensal microbiota are required for systemic inflammation triggered by necrotic dendritic cells. - Cell reports
The relationship between dendritic cells (DCs) and commensal microflora in shaping systemic immune responses is not well understood. Here, we report that mice deficient for the Fas-associated death domain in DCs developed systemic inflammation associated with elevated proinflammatory cytokines and increased myeloid and B cells. These mice exhibited reduced DCs in gut-associated lymphoid tissues due to RIP3-dependent necroptosis, whereas DC functions remained intact. Induction of systemic inflammation required DC necroptosis and commensal microbiota signals that activated MyD88-dependent pathways in other cell types. Systemic inflammation was abrogated with the administration of broad-spectrum antibiotics or complete, but not DC-specific, deletion of MyD88. Thus, we have identified a previously unappreciated role for commensal microbiota in priming immune cells for inflammatory responses against necrotic cells. These studies demonstrate the impact intestinal microflora have on the immune system and their role in eliciting proper immune responses to harmful stimuli.Copyright Â© 2013 The Authors. Published by Elsevier Inc. All rights reserved.
Factors predicting patient perception of dysphonia caused by benign vocal fold lesions. - The Laryngoscope
To assess factors that may be predictive of patient perception of dysphonia severity, as quantified by the Voice Handicap Index (VHI) score. We hypothesize that 1) level of vocal demand; 2) auditory-perceptual evaluation of dysphonia severity; and 3) vocal function, as defined by phonatory glottal closure and mucosal wave vibration, are the most significant predictors of VHI score.: Retrospective review of 100 patients with benign vocal fold lesions.Variables assessed for predictive value to VHI score are level of vocal demands, auditory-perceptual evaluation of dysphonia severity, integrity of mucosal wave vibration and phonatory glottal closure, lesion type, duration of current complaint, smoking, age, and sex. Harmonic to noise ratio was assessed in a subset of 50 patients.Patients with routine voice use had significantly lower VHI scores than those with more intensive (nonsinging/acting) vocal demands. Patients who quit smoking had greater VHI scores than those who currently smoke or never started. Patients with long-standing dysphonia tended to have lower VHI scores than those with shorter duration vocal complaints. Auditory-perceptual assessment of dysphonia severity and harmonic to noise ratio were weak predictors of VHI score. Age, sex, lesion type, phonatory glottal closure, and mucosal wave vibration were not significant predictors of VHI score.Patient perception of dysphonia severity is independent of many factors commonly assessed during the evaluation of voice disorders. It appears to be an important independent element in the assessment of the effect of a benign vocal fold lesion and critical to therapeutic decision-making.
Map & Directions
128 Mott St # 138 Suite 608 New York, NY 10013
168 Canal St 4Th Floor