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Dr. Julie  Young Campbell  Od image

Dr. Julie Young Campbell Od

725 N Tyndall Pkwy
Panama City FL 32404
850 853-3426
Medical School: University Alabama Birmingham - School Of Optometry - 1994
Accepts Medicare: Yes
Participates In eRX: No
Participates In PQRS: No
Participates In EHR: No
License #: OPC2920
NPI: 1669510111
Taxonomy Codes:
152W00000X

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Awards & Recognitions

About Us

Practice Philosophy

Conditions

Dr. Julie Young Campbell is associated with these group practices

Procedure Pricing

HCPCS Code Description Average Price Average Price
Allowed By Medicare
HCPCS Code:92004 Description:Eye exam new patient Average Price:$75.00 Average Price Allowed
By Medicare:
$75.00
HCPCS Code:99213 Description:Office/outpatient visit est Average Price:$56.00 Average Price Allowed
By Medicare:
$56.00
HCPCS Code:76514 Description:Echo exam of eye thickness Average Price:$12.00 Average Price Allowed
By Medicare:
$12.00
HCPCS Code:66984 Description:Cataract surg w/iol 1 stage Average Price:$130.00 Average Price Allowed
By Medicare:
$130.00
HCPCS Code:92014 Description:Eye exam & treatment Average Price:$75.00 Average Price Allowed
By Medicare:
$75.00
HCPCS Code:92083 Description:Visual field examination(s) Average Price:$60.00 Average Price Allowed
By Medicare:
$60.00

HCPCS Code Definitions

92004
Ophthalmological services: medical examination and evaluation with initiation of diagnostic and treatment program; comprehensive, new patient, 1 or more visits
99213
Office or other outpatient visit for the evaluation and management of an established patient, which requires at least 2 of these 3 key components: An expanded problem focused history; An expanded problem focused examination; Medical decision making of low complexity. Counseling and coordination of care with other physicians, other qualified health care professionals, or agencies are provided consistent with the nature of the problem(s) and the patient's and/or family's needs. Usually, the presenting problem(s) are of low to moderate severity. Typically, 15 minutes are spent face-to-face with the patient and/or family.
76514
Ophthalmic ultrasound, diagnostic; corneal pachymetry, unilateral or bilateral (determination of corneal thickness)
66984
Extracapsular cataract removal with insertion of intraocular lens prosthesis (1 stage procedure), manual or mechanical technique (eg, irrigation and aspiration or phacoemulsification)
92014
Ophthalmological services: medical examination and evaluation, with initiation or continuation of diagnostic and treatment program; comprehensive, established patient, 1 or more visits
92083
Visual field examination, unilateral or bilateral, with interpretation and report; extended examination (eg, Goldmann visual fields with at least 3 isopters plotted and static determination within the central 30°, or quantitative, automated threshold perimetry, Octopus program G-1, 32 or 42, Humphrey visual field analyzer full threshold programs 30-2, 24-2, or 30/60-2)

Medical Malpractice Cases

None Found

Medical Board Sanctions

None Found

Referrals

NPI
Doctor Name
Specialty
Count
1366472383
Ophthalmology
285
1811041569
General Practice
253
1659345114
Internal Medicine
165
1871522201
Pulmonary Disease
136
1912967142
Neurology
127
1922027515
Cardiovascular Disease (Cardiology)
121
1235197518
Cardiovascular Disease (Cardiology)
120
1720085004
Diagnostic Radiology
114
1801804117
Internal Medicine
112
1073562260
Cardiovascular Disease (Cardiology)
112
*These referrals represent the top 10 that Dr. Young Campbell has made to other doctors

Publications

Effects of estrogen on stress-induced premature senescence of vascular smooth muscle cells: a novel mechanism for the "time window theory" of menopausal hormone therapy. - Atherosclerosis
To investigate the effects of estrogen on stress-induced premature senescence of vascular smooth muscle cells (VSMCs) and the underlying mechanisms.VSMCs of passage 2-3 cultured from young (2 months) and old (18 months) female SD rats were induced into premature senescence by exposure to 150 μmol/L H(2)O(2) in the presence or absence of different concentrations of 17β-estradiol (E(2)). The expression or activation of senescence-associated beta-galactosidase (SA-β-Gal), DcR2, oncogene Ras, p38, PRAK, p53, p21, p16 and Rb was detected by flow cytometry, pull-down assay or Western blot.Flow cytometry analysis showed that in the VSMCs from young rats pre-administration of E(2) significantly suppressed the H(2)O(2)-induced premature senescence (reducing both percentage of SA-β-Gal positive cells and cellular expression of DcR2) in a dose-dependent manner; these senescent-inhibiting effects of E(2) could be blocked by an estrogen receptor antagonist ICI 182,780 (10(-5)mol/L). Pull-down assay or Western blot analysis revealed that pre-administration of 10(-8)mol/L E(2) significantly reduced the H(2)O(2)-induced activation of oncogene Ras, as well as activity of p16 and p38 MAPK, and expression of PRAK, p53, p21 and p-Rb. Unexpectedly, in the VSMCs from old rats the senescent-inhibiting effect of E(2) disappeared and switched to a senescent-promoting action at 10(-8)mol/L. This senescent-promoting effect could be enhanced by ICI 182,780 and eliminated by a cytochrome P450s inhibitor ABT.Estrogen inhibits stress-induced premature senescence of VSMCs from young female through its receptor-mediated suppression of both Ras-p38-PRAK-p53-p21-Rb and Ras-p16-Rb pathways, but this effect disappeared and even more switched to a senescent-promoting action in the cells from old body probably due to a side effect of estrogen metabolites.Copyright © 2011 Elsevier Ireland Ltd. All rights reserved.

Map & Directions

725 N Tyndall Pkwy Panama City, FL 32404
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